Geriatric Healthcare Specialist Whiteville NC

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Nicole A Collins
(919) 496-4250
205 Sandalwood Ave
Louisburg, NC
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
James Sheridan Parsons
(919) 832-5125
704 W Jones St
Raleigh, NC
Specialty
General Practice, Geriatric Medicine

Data Provided by:
Jan Busby Whitehead, MD
(919) 966-2276
Cb7550
Chapel Hill, NC
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Tx Med Branch Galveston, Galveston Tx 77550
Graduation Year: 1977
Hospital
Hospital: University Of North Carolina H, Chapel Hill, Nc
Group Practice: Unc Dept-Internal Medicine

Data Provided by:
Katja Ingrid Elbert Avila, MD
Duke Univ Med Ctr
Durham, NC
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Mi Med Sch, Ann Arbor Mi 48109
Graduation Year: 1999

Data Provided by:
Marco Pahor, MD
(336) 713-8520
1 Medical Center Blvd,
Winston Salem, NC
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Cattol De Sacro Cuore, Fac Di Med E Chirurgia, Roma, Italy
Graduation Year: 1980

Data Provided by:
Lea Corinne Watson, MD
(919) 966-4131
201 Old Larkspur Way
Chapel Hill, NC
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Va Commonwealth Univ, Med Coll Of Va Sch Of Med, Richmond Va 23298
Graduation Year: 1997

Data Provided by:
Charles Davant
(828) 295-3116
366 Chestnut Dr
Blowing Rock, NC
Specialty
Family Practice, Geriatric Medicine, Emergency Medicine

Data Provided by:
Mitchell Tod Heflin, MD
(919) 660-7561
101 Polo Pl
Rougemont, NC
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Va Sch Of Med, Charlottesville Va 22908
Graduation Year: 1994

Data Provided by:
Sylvie Bastajian, MD
(704) 783-2025
5904 Daria Ct
Charlotte, NC
Specialties
Internal Medicine, Geriatrics
Gender
Male
Education
Medical School: American Univ Of Beirut, Fac Of Med, Bei
Graduation Year: 1990

Data Provided by:
Derek M Reed
(704) 263-0300
159 E Dallas Rd
Stanley, NC
Specialty
Family Practice, Geriatric Medicine

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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