Geriatric Healthcare Specialist Whiteville NC

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Kesi Tabia Kindle, MD
(252) 816-4100
4975 Seward Rd
Pfafftown, NC
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Howard Univ Coll Of Med, Washington Dc 20059
Graduation Year: 1999

Data Provided by:
Gayani Yahampath Dasanayaka
(336) 544-5400
1309 N Elm Street
Greensboro, NC
Specialty
Geriatric Medicine

Data Provided by:
Heidi White
(919) 620-4467
2100 Erwin Rd
Durham, NC
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Cathleen S Colon Emeric, MD
4220 N Roxboro St
Durham, NC
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Johns Hopkins Univ Sch Of Med, Baltimore Md 21205
Graduation Year: 1994

Data Provided by:
Jan Busby Whitehead, MD
(919) 966-2276
Cb7550
Chapel Hill, NC
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Tx Med Branch Galveston, Galveston Tx 77550
Graduation Year: 1977
Hospital
Hospital: University Of North Carolina H, Chapel Hill, Nc
Group Practice: Unc Dept-Internal Medicine

Data Provided by:
Elizabeth J Herskovits Castillo
(828) 257-4730
118 Wt Weaver Blvd
Asheville, NC
Specialty
Geriatric Medicine

Data Provided by:
Nancy Carolyn Lehman, MD
(828) 252-0015
14 S Pack Sq Ste 362
Asheville, NC
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Med Univ Of Sc Coll Of Med, Charleston Sc 29425
Graduation Year: 1996

Data Provided by:
M C Deirdre Johnston, MD
(336) 716-9687
8th Flr Csb Dept Pathol
Winston Salem, NC
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Univ Coll Dublin, Nat'L Univ Of Ireland, Fac Of Med, Dublin
Graduation Year: 1979

Data Provided by:
Syed Taj-Ul Arfeen, MD
(336) 832-9800
515 Hannah McKenzie Dr
Greensboro, NC
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Dow Med Coll, Univ Of Karachi, Karachi, Pakistan
Graduation Year: 1990

Data Provided by:
Naveena Polavarapu, MD
(847) 723-2210
Raleigh, NC
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Siddartha Med Coll, Univ Of Hlth Sci, Vijayawada, Ap, India
Graduation Year: 1997

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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