Geriatric Healthcare Specialist Wallingford CT

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Maria Ditomasso, MD
(203) 573-6525
17 Penny Ln
Wallingford, CT
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Mc Gill Univ, Fac Of Med, Montreal, Que, Canada
Graduation Year: 1990

Data Provided by:
Audrey Lefkowitz
(203) 235-5445
469 E Main Street
Meriden, CT
Specialty
Geriatric Medicine

Data Provided by:
David Mark Taraskevich
(203) 237-2200
237 Liberty St
Meriden, CT
Specialty
Family Practice, Geriatric Medicine

Data Provided by:
Alan Paul Siegal, MD
(203) 288-0414
60 Washington Ave Ste 203
Hamden, CT
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Univ Of Louisville Sch Of Med, Louisville Ky 40202
Graduation Year: 1979
Hospital
Hospital: Yale -New Haven Hosp, New Haven, Ct; St Raphaels Hosp, New Haven, Ct
Group Practice: Geriatric & Adult Psychiatry

Data Provided by:
Matthew Jay Raider, MD
(860) 344-5920
540 Saybrook Rd
Middletown, CT
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Mi Med Sch, Ann Arbor Mi 48109
Graduation Year: 1979

Data Provided by:
Patrick John Berger, DO
22 Masonic Ave
Wallingford, CT
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Des Moines Univ, Coll Osteo Med & Surg, Des Moines Ia 50312
Graduation Year: 1992

Data Provided by:
Audrey Lefkowitz, MD
(203) 235-5445
469 E Main St
Meriden, CT
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Auto De Guadalajara, Fac De Med, Guadalajara, Jalisco, Mexico
Graduation Year: 1976

Data Provided by:
Neil Scollan
(203) 235-5445
469 E Main Street
Meriden, CT
Specialty
Geriatric Medicine

Data Provided by:
Jeanne Marie Jackson, MD
(203) 288-0414
60 Washington Ave Ste 203
Hamden, CT
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Or Hlth Sci Univ Sch Of Med, Portland Or 97201
Graduation Year: 1989

Data Provided by:
Glendo Lim Tangarorang, MD
(203) 407-8956
51 Kaye Vue Dr Apt D
Hamden, CT
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Cebu Inst Of Med, Cebu City, Philippines
Graduation Year: 1990

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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