Geriatric Healthcare Specialist South Sioux City NE

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

William Lee Lyons, MD
(415) 514-0718
981320 Nebraska Medical Ctr
Omaha, NE
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Washington Univ Sch Of Med, St Louis Mo 63110
Graduation Year: 1995

Data Provided by:
Rebecca B Reilly
(402) 354-3152
8303 Dodge St
Omaha, NE
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Fred Harvey Ayers, MD
(402) 773-5553
PO Box 601
Sutton, NE
Specialties
Geriatrics, Geriatric Medicine-Family Practice, Psychoanalysis
Gender
Male
Education
Medical School: Wayne State Univ Sch Of Med, Detroit Mi 48201
Graduation Year: 1972
Hospital
Hospital: Mary Lanning Memorial Hospital, Hastings, Ne
Group Practice: Sutton Medical Clinic

Data Provided by:
Rebecca Bohi Reilly, MD
(402) 496-2800
8303 Dodge St
Omaha, NE
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Ne Coll Of Med, Omaha Ne 68198
Graduation Year: 1985
Hospital
Hospital: Nebraska Methodist Hospital, Omaha, Ne
Group Practice: Physicians Clinic Methodist Health West

Data Provided by:
Thomas S Robertson
(402) 727-5200
2350 N Clarkson St
Fremont, NE
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Mary A Wampler
(402) 559-9800
988095 Nebraska Medical Ctr
Omaha, NE
Specialty
Geriatric Medicine

Data Provided by:
Mark Allen Tompkins
(308) 385-6252
2300 W Capital Ave
Grand Island, NE
Specialty
Geriatric Medicine

Data Provided by:
Arash Nassim
(308) 262-1755
1320 S St
Bridgeport, NE
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Abelardo Cruz, MD
(402) 894-1431
19814 Elkhorn Ridge Dr
Elkhorn, NE
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine, Rheumatology
Gender
Male
Education
Medical School: Univ Of The Philippines, Coll Of Med, Manila, Philippines
Graduation Year: 1993
Hospital
Hospital: Nebraska Methodist Hospital, Omaha, Ne
Group Practice: Physicians Clinic

Data Provided by:
Julie Kay Filips, MD
(402) 474-1511
7547 Brummond Dr
Lincoln, NE
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Univ Of Sd Sch Of Med, Vermillion Sd, 57069
Graduation Year: 1997

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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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