Geriatric Healthcare Specialist Searcy AR

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Eugene Allen Joseph, MD
(501) 268-7143
1304 S Main St
Searcy, AR
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Univ Of Ar Coll Of Med, Little Rock Ar 72205
Graduation Year: 1961

Data Provided by:
Noel Melendres Medel, MD
(501) 279-7077
403 S Poplar St Ste E
Searcy, AR
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Santo Tomas, Fac Of Med And Surg, Manila, Philippines
Graduation Year: 1981

Data Provided by:
Joseph Dale Sarnicki
(870) 798-4299
402 Lee St
Hampton, AR
Specialty
Geriatric Medicine

Data Provided by:
James Russell Lambert, MD
(817) 924-2818
PO Box 476
Greenbrier, AR
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Univ Of Tx Southwestern Med Ctr At Dallas, Med Sch, Dallas Tx 75235
Graduation Year: 1960
Hospital
Hospital: Baylor All Saints Med Ctr -Fo, Fort Worth, Tx

Data Provided by:
Prabhat K Hebbar
(501) 833-8400
2215 Wildwood Ave
Sherwood, AR
Specialty
Family Practice, Internal Medicine, Geriatric Medicine

Data Provided by:
Eugene Allen Joseph, MD
(501) 268-7143
1304 S Main St
Searcy, AR
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Univ Of Ar Coll Of Med, Little Rock Ar 72205
Graduation Year: 1961

Data Provided by:
Kathryn I Reeder, MD
(501) 327-5440
32 Military Ridge Rd
Conway, AR
Specialties
Internal Medicine, Geriatrics
Gender
Male
Education
Medical School: Univ Of Ar Coll Of Med, Little Rock Ar 7
Graduation Year: 1946

Data Provided by:
Timothy Scott Simmons
(501) 686-8000
4301 W Markham St # 783
Little Rock, AR
Specialty
Geriatric Medicine

Data Provided by:
Robert Lawrence Stanley
(501) 833-8400
2215 Wildwood Ave
Sherwood, AR
Specialty
Family Practice, Internal Medicine, Geriatric Medicine

Data Provided by:
Kanthi Charan Narra, MD
(501) 526-6547
Univ of Arkansas for Medical Sciences Reynolds Institute on Aging 629 Jac
Little Rock, AR
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Graduation Year: 2003

Data Provided by:
Data Provided by:

Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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