Geriatric Healthcare Specialist Scottsboro AL

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Nageswara Rao Chava, MD
(334) 272-4670
6524 Eastwood Glen Dr
Montgomery, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Sri Venkatesvara Med Coll, Univ Hlth Sci, Tirupati, Ap, India
Graduation Year: 1972

Data Provided by:
Ritky C Dy
(205) 280-6789
1221 7th St S
Clanton, AL
Specialty
Family Practice, Geriatric Medicine

Data Provided by:
Sabin Sebastian, MD
(205) 835-2031
220 Eden View Dr
Birmingham, AL
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Kasturba Med Coll, Mysore Univ, Mangalore, Karnataka, India
Graduation Year: 1992

Data Provided by:
Christine Seel Ritchie, MD
619 South 19th Street
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Nc At Chapel Hill Sch Of Med, Chapel Hill Nc 27599
Graduation Year: 1988

Data Provided by:
Avinash Arjun Nichani, MD
Tuscaloosa, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Dr D Y Patil Med Coll, Shivaji Univ, Kolhapur, Maharashtra
Graduation Year: 1995

Data Provided by:
Mitchell Lee Brown, MD
(205) 934-0820
306 Lyons Harrison Research Bldg,
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Ross Univ, Sch Of Med & Vet Med, Roseau, Dominica
Graduation Year: 1987

Data Provided by:
Igor Bidikov
(256) 463-7454
40 Giles St
Heflin, AL
Specialty
Geriatric Medicine

Data Provided by:
Gary Lee Howard, MD
(205) 655-2002
33 Salem Rd
Montevallo, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine, General Practice
Gender
Male
Education
Medical School: Univ Of Al Sch Of Med, Birmingham Al 35294
Graduation Year: 1984
Hospital
Hospital: Baptist Shelby Med Ctr, Alabaster, Al
Group Practice: Calera Family Health Ctr

Data Provided by:
Stephen Brown Henderson
(256) 582-2581
2308 Homer Clayton Dr
Guntersville, AL
Specialty
Family Practice, Geriatric Medicine, Sports Medicine

Data Provided by:
Shyla Reddy
(251) 434-3475
1504 Springhill Ave
Mobile, AL
Specialty
Family Practice, Geriatric Medicine

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Food for Thought

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By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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