Geriatric Healthcare Specialist Santa Maria CA

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Michael K Kazak
(805) 922-8269
1040 W Main St
Santa Maria, CA
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Huma Zahid Khan, MD
Gardena Medicine Office B
Gardena, CA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Sind Med Coll, Univ Of Karachi, Karachi, Pakistan
Graduation Year: 1993

Data Provided by:
Serge Alain Lindner, MD
801 Ygnacio Valley Rd
Walnut Creek, CA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Wa Sch Of Med, Seattle Wa 98195
Graduation Year: 1998

Data Provided by:
Sudhir Prasad Kakarla, MD
152 Pioneer Ln
Bishop, CA
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Seth G S Med Coll, Univ Of Bombay, Bombay, Maharashtra, India
Graduation Year: 1997

Data Provided by:
Elizabeth S Menkin, MD
(408) 972-3000
260 International Cir
San Jose, CA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Ca, San Francisco, Sch Of Med, San Francisco Ca 94143
Graduation Year: 1981

Data Provided by:
Linda Ann Morrow, MD
(510) 593-4814
2600 N St Apt 6
Sacramento, CA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Med Coll Of Pa, Philadelphia Pa 19129
Graduation Year: 1980

Data Provided by:
Kenneth K Tram
(714) 964-6229
9930 Talbert Ave
Fountain Valley, CA
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Louise Christie Walter, MD
415-221-4810 x3052
6 Locksley Ave Apt 2L
San Francisco, CA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Stanford Univ Sch Of Med, Stanford Ca 94305
Graduation Year: 1995

Data Provided by:
Daniel Lewis Gornel, MD
(310) 247-7700
1136 Hi Point St
Los Angeles, CA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Harvard Med Sch, Boston Ma 02115
Graduation Year: 1957

Data Provided by:
Erik Paul Schten
(415) 448-1555
400 Professional Center Dr
Novato, CA
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Data Provided by:

Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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