Geriatric Healthcare Specialist Pinson AL

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Madison Bates Redwine, MD
(205) 592-1900
1007 42nd St S
Birmingham, AL
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Med Coll Of Ga Sch Of Med, Augusta Ga 30912
Graduation Year: 1994

Data Provided by:
Cynthia Holcomb Baker, MD
619 19th St S
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of South Al Coll Of Med, Mobile Al 36688
Graduation Year: 1996

Data Provided by:
Charlotte Jane Williams, MD
2000 6th Ave S
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Sheffield Med Sch, Fac Med/Dent, Sheffield (352-10 Pr 1/71)
Graduation Year: 1996

Data Provided by:
Paul Douglas Roller, MD
2000 6th Ave S # F
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Va Commonwealth Univ, Med Coll Of Va Sch Of Med, Richmond Va 23298
Graduation Year: 1991

Data Provided by:
J Walden Retan
(205) 967-2900
3105 Warrington Rd
Mountain Brook, AL
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Mary Margaret O'Donoghue, MD
3146 Pine Ridge Rd
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Coll Of Galway, Nat'L Univ Of Ireland, Fac Of Med, Galway
Graduation Year: 1980

Data Provided by:
Christine Seel Ritchie, MD
619 South 19th Street
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Nc At Chapel Hill Sch Of Med, Chapel Hill Nc 27599
Graduation Year: 1988

Data Provided by:
Terri Sue Steele, MD
(205) 934-6054
1713 6th Ave S
Birmingham, AL
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Univ Of South Al Coll Of Med, Mobile Al 36688
Graduation Year: 1986

Data Provided by:
Mitchell Lee Brown, MD
(205) 934-0820
306 Lyons Harrison Research Bldg,
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Ross Univ, Sch Of Med & Vet Med, Roseau, Dominica
Graduation Year: 1987

Data Provided by:
Marisol S Sanchez Lance, DO
(205) 423-0597
3532 Victoria Rd
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Kirksville Coll Of Osteo Med, Kirksville Mo 63501
Graduation Year: 1994

Data Provided by:
Data Provided by:

Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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