Geriatric Healthcare Specialist Norwalk CT

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Malcolm Gordon, MD
(203) 858-2482
38 Harbor View Ave
Norwalk, CT
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Boston Univ Sch Of Med, Boston Ma 02118
Graduation Year: 1948
Hospital
Hospital: Norwalk Hosp, Norwalk, Ct
Group Practice: Geriatric Psychiatry Assoc

Data Provided by:
Neil P Dolan, MD
(203) 655-1559
10 Corbin Dr
Darien, CT
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Univ Coll Dublin, Nat'L Univ Of Ireland, Fac Of Med, Dublin
Graduation Year: 1989

Data Provided by:
Barney S Spivack, MD
(203) 594-5304
105 Long Lots Rd
Westport, CT
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Mt Sinai Sch Of Med Of The City Univ Of Ny, New York Ny 10029
Graduation Year: 1978

Data Provided by:
Marina L Blagodatny, MD
100 Cedar Heights Rd
Stamford, CT
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Second Moscow Med Inst, Russian State Med Univ, Moscow, Russia
Graduation Year: 1981

Data Provided by:
Steven H Kunkes
(203) 292-2000
1305 Post Rd
Fairfield, CT
Specialty
Cardiology, Internal Medicine, Geriatric Medicine, Cardiovascular Disease

Data Provided by:
Randi Diamond
(203) 838-7400
128 East Ave
Norwalk, CT
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Sudhakar Subbanna Shetty, MD
10 Corbin Dr
Darien, CT
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Mr Med Coll, Gulbarga Univ, Gulbarga, Karnataka, India
Graduation Year: 1986

Data Provided by:
Anri Kissilenko, MD
208 Valley Rd
New Canaan, CT
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Sofia Med Academy, Fac Of Med, Sofia, Bulgaria
Graduation Year: 1990

Data Provided by:
Walter Michael Kobialka
(203) 325-0764
1275 Summer St Ste 102
Stamford, CT
Specialty
Geriatric Medicine

Data Provided by:
James Michael Luciano, MD
(914) 260-4861
121 Brook Farm Rd E
Pound Ridge, NY
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Suny-Hlth Sci Ctr At Brooklyn, Coll Of Med, Brooklyn Ny 11203
Graduation Year: 1982

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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