Geriatric Healthcare Specialist Milford MA

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Eugene Charles Jagella, MD
(508) 529-8101
10 Veterans Memorial Dr
Milford, MA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Med Coll Of Pa, Philadelphia Pa 19129
Graduation Year: 1986

Data Provided by:
Sharon C Eypper, MD
(508) 856-4977
2 Thomas Rice Dr
Westborough, MA
Specialties
Internal Medicine, Geriatrics
Gender
Male
Education
Medical School: Univ Of Mi Med Sch, Ann Arbor Mi 48109
Graduation Year: 1982

Data Provided by:
James David Burrill
(401) 769-0800
560 Cumberland Hill Rd
Woonsocket, RI
Specialty
Geriatric Medicine

Data Provided by:
Robert Scott Keyes, MD
39 Keyes House Rd
Shrewsbury, MA
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Univ Of Cincinnati Coll Of Med, Cincinnati Oh 45267
Graduation Year: 2000

Data Provided by:
Walter H Ettinger, MD
(917) 463-9855
55 Lake Ave N Rm H1-773
Worcester, MA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Johns Hopkins Univ Sch Of Med, Baltimore Md 21205
Graduation Year: 1978

Data Provided by:
Gary Stuart Moak, MD
(508) 366-2106
21 Longmeadow Rd
Westborough, MA
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Umdnj-Robt W Johnson Med Sch, New Brunswick Nj 08901
Graduation Year: 1982
Hospital
Hospital: St Vincent Hospital -Vernon H, Worcester, Ma; Whittier Rehabilitation Hosp, Haverhill, Ma
Group Practice: Umass Memorial Medical Group

Data Provided by:
Linyuh Lee, MD
(508) 528-8015
102 King St
Norfolk, MA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Boston Univ Sch Of Med, Boston Ma 02118
Graduation Year: 1989
Hospital
Hospital: Beth Israel Deaconess Med Ctr, Boston, Ma
Group Practice: South Cove Comm Hlth Ctr

Data Provided by:
Avanish Mehta, MD
(401) 767-2500
20 Cumberland Hill Rd Unit 105
Woonsocket, RI
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Maulana Azad Med Coll, Univ Of Delhi, New Delhi, Delhi, India
Graduation Year: 1975

Data Provided by:
Vinay Kumar
(508) 460-3872
126 Union Street
Marlborough, MA
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Robert A Witzburg, MD
(617) 638-4630
10 Berkeley Rd
Wellesley, MA
Specialties
Internal Medicine, Geriatrics
Gender
Male
Education
Medical School: Boston Univ Sch Of Med, Boston Ma 02118
Graduation Year: 1977

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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