Geriatric Healthcare Specialist Madison WI

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Muhammad Khwaja Mansoor Ahmed, MD
Madison, WI
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Aga Khan Med Coll, Aga Khan Univ, Karachi, Pakistan
Graduation Year: 1995

Data Provided by:
Mary L Carnes
(608) 263-7740
600 Highland Ave
Madison, WI
Specialty
Geriatric Medicine

Data Provided by:
Lora Lee Wiggins, MD
600 Highland Ave
Madison, WI
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Suny-Hlth Sci Ctr At Brooklyn, Coll Of Med, Brooklyn Ny 11203
Graduation Year: 1987

Data Provided by:
Joseph P Connor
(608) 265-1700
600 Highland Ave
Madison, WI
Specialty
Geriatric Medicine

Data Provided by:
Michael Joseph La Breche, MD
Madison, WI
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Med Coll Of Wi, Milwaukee Wi 53226
Graduation Year: 1965

Data Provided by:
Richard Merrit Reich, MD
(608) 287-2435
20 S Park St
Madison, WI
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Cornell Univ Med Coll, New York Ny 10021
Graduation Year: 1975

Data Provided by:
Rina Ankur Patel, MD
(608) 263-8500
600 Highland Ave # K4/B100
Madison, WI
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Ms Ramaiah Med Coll, Bangalore Univ, Bangalore, Karnataka, India
Graduation Year: 1989

Data Provided by:
Cynthia M Carlsson
(608) 262-8597
600 Highland Ave
Madison, WI
Specialty
Geriatric Medicine

Data Provided by:
Jeanne Marie Howard, MD
(608) 274-4190
1126 Sunridge Dr
Madison, WI
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Med Coll Of Wi, Milwaukee Wi 53226
Graduation Year: 1994

Data Provided by:
Steven Robt Barczi, MD
(608) 262-7000
409 Holly Ave
Madison, WI
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine, Sleep Medicine
Gender
Male
Education
Medical School: Univ Of Il Coll Of Med, Chicago Il 60680
Graduation Year: 1989

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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