Geriatric Healthcare Specialist Madison AL

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Anupama Dronavalli Yedla, MD
301 Governors Dr SE
Huntsville, AL
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Dr Br Ambedkar Med Coll, Bangalore Univ, Bangalore, Karnataka
Graduation Year: 1994

Data Provided by:
Lisa Philippart
(256) 665-3607
Huntsville, AL
Practice Areas
Eating Disorders, Aging/Gerontological, Depression/Grief/Chronically or Terminally Ill, Mental Health/Agency Counseling
Certifications
National Certified Counselor

Wayde Zane Slocumb
(256) 797-6095
Huntsville, AL
Practice Areas
Childhood & Adolescence, Aging/Gerontological, Couples & Family, Depression/Grief/Chronically or Terminally Ill
Certifications
National Certified Counselor

Manivannan Neelamegam, MD
Tuscaloosa, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Thanjavur Med Coll, Dr M G R Med Univ, Thanjavur, Tn, India
Graduation Year: 1978

Data Provided by:
Jaehneth Ann Ewell-Taylor
(334) 727-0550
2400 Hospital Rd
Tuskegee, AL
Specialty
Geriatric Medicine

Data Provided by:
Laura Elaine Dyer, MD
(256) 536-5635
4845 Cove Creek Dr SE
Brownsboro, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Al Sch Of Med, Birmingham Al 35294
Graduation Year: 1998

Data Provided by:
Leslie Powell
(256) 260-7334
Decatur, AL
Practice Areas
Childhood & Adolescence, Aging/Gerontological, Depression/Grief/Chronically or Terminally Ill, Mental Health/Agency Counseling, Supervision
Certifications
National Certified Counselor

Gary Lee Howard, MD
(205) 655-2002
33 Salem Rd
Montevallo, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine, General Practice
Gender
Male
Education
Medical School: Univ Of Al Sch Of Med, Birmingham Al 35294
Graduation Year: 1984
Hospital
Hospital: Baptist Shelby Med Ctr, Alabaster, Al
Group Practice: Calera Family Health Ctr

Data Provided by:
Jesus Ramon Rodriguez, MD
(305) 534-9009
170 Pine Ct
Pell City, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Miami Sch Of Med, Miami Fl 33101
Graduation Year: 1985

Data Provided by:
Mary Margaret O'Donoghue, MD
3146 Pine Ridge Rd
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Coll Of Galway, Nat'L Univ Of Ireland, Fac Of Med, Galway
Graduation Year: 1980

Data Provided by:
Data Provided by:

Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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