Geriatric Healthcare Specialist Lake Havasu City AZ

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Ruth Post
(623) 247-6266
4616 N 51st Ave
Phoenix, AZ
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Enoch Arenas Barrios, MD
Yuma, AZ
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Univ Nacl Auto De Mexico, Fac De Med, Mexico Df, Mexico
Graduation Year: 1985

Data Provided by:
Louis G Greco
(480) 985-1093
55 S 63rd St Ste 6
Mesa, AZ
Specialty
Geriatric Medicine

Data Provided by:
Arlene V De Guzman, MD
10401 W Thunderbird Blvd
Sun City, AZ
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Female
Education
Medical School: Fatima Coll Of Med, Valenzuela, Manila, Philippines
Graduation Year: 1991

Data Provided by:
Alfredo Lim, MD
(602) 439-1200
1214 W Hayward Ave
Phoenix, AZ
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Far Eastern Univ, Dr N Reyes Med Fndn Inst Of Med, Manila, Philippines
Graduation Year: 1986

Data Provided by:
Gary Howard Salzman, MD
(602) 239-5277
5402 E Cholla St
Scottsdale, AZ
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Rush Med Coll Of Rush Univ, Chicago Il 60612
Graduation Year: 1977

Data Provided by:
Ma Lucia Magat Gregorio, MD
(623) 977-7201
Phoenix, AZ
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of The East, Ramon Magsaysay Mem Med Ctr, Quezon City
Graduation Year: 1988

Data Provided by:
Melvin Hector
(520) 615-6200
1775 E Skyline Dr
Tucson, AZ
Specialty
Family Practice, Geriatric Medicine

Data Provided by:
Jane L De Beer Buehrer, MD
(602) 681-8762
5635 E Lincoln Dr Ste 29
Paradise Valley, AZ
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Suny-Hlth Sci Ctr At Brooklyn, Coll Of Med, Brooklyn Ny 11203
Graduation Year: 1952

Data Provided by:
Bonnie Ann Goodman, DO
4600 S Park Ave Ste 5
Tucson, AZ
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Female
Education
Medical School: Chicago Coll Of Osteo Med, Midwestern Univ, Chicago Il 60615
Graduation Year: 1978

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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