Geriatric Healthcare Specialist Johnson City TN

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Stephanie C Leeper, MD
(423) 439-2036
PO Box 70580
Johnson City, TN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: E Tn State Univ J H Quillen Coll Of Med, Johnson City Tn 37614
Graduation Year: 1987
Hospital
Hospital: Johnson City Med Ctr, Johnson City, Tn
Group Practice: Etsu Physicians & Assoc

Data Provided by:
Craig Nathaniel Moss, MD
PO Box 70578etsu
Johnson City, TN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: New York Med Coll, Valhalla Ny 10595
Graduation Year: 1980

Data Provided by:
Ronald C Hamdy
(423) 439-7280
325 N State Of Franklin Rd
Johnson City, TN
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Rodney L Holladay
(423) 282-1480
119 Boone Ridge Dr
Johnson City, TN
Specialty
Geriatric Medicine

Data Provided by:
David Harold Franzus, MD
(423) 247-3717
202 W Ravine Rd
Kingsport, TN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: E Tn State Univ J H Quillen Coll Of Med, Johnson City Tn 37614
Graduation Year: 1983

Data Provided by:
Ivan Merkelj, MD
Johnson City, TN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Nac Federico Villarreal, Prog Acad De Med Humana, Lima, Peru
Graduation Year: 1986

Data Provided by:
Kenneth E Olive
(423) 439-7280
325 N State Of Franklin Rd
Johnson City, TN
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Ronald Charles Hamdy, MD
(423) 439-8830
325 N State of Franklin Rd # F
Johnson City, TN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Alexandria, Fac Of Med, Alexandria, Egypt (330-03 Pr 1/71)
Graduation Year: 1968

Data Provided by:
Christine S Barber, MD
(423) 538-7659
120 River Bluff Way
Bluff City, TN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Southern Ca Sch Of Med, Los Angeles Ca 90033
Graduation Year: 1993

Data Provided by:
Dean Mc Laughlin
2408 Susannah St
Johnson City, TN
Specialty
Geriatric Family Practice, Alzheimer's Specialist

Data Provided by:

Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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