Geriatric Healthcare Specialist Jackson MS

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Holland M Addison
(601) 352-2273
501 Marshall St
Jackson, MS
Specialty
Geriatric Medicine

Data Provided by:
Kimberly C Teal
(601) 984-6197
2500 North State Street
Jackson, MS
Specialty
Geriatric Medicine

Data Provided by:
Noella A Misquita, MD
(410) 876-9680
Jackson, MS
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Dublin, Trinity Coll, Sch Of Physic, Dublin, Ireland
Graduation Year: 1986

Data Provided by:
Zeba Iqbal, MD
Ridgeland, MS
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Aga Khan Med Coll, Aga Khan Univ, Karachi, Pakistan
Graduation Year: 1994

Data Provided by:
Clinton Wallace
(601) 354-1234
1421 N State St
Jackson, MS
Specialty
Geriatric Internal Medicine, Alzheimer's Specialist

Chere Hunter Peel
(601) 362-4471
1500 E Woodrow Wilson Ave
Jackson, MS
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Katharine H Travis Gregg, MD
(601) 984-6440
2500 N State St
Jackson, MS
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Ms Sch Of Med, Jackson Ms 39216
Graduation Year: 1994
Hospital
Hospital: M S Methodist Rehab Center, Jackson, Ms; Univ Of Mississippi Med Ctr, Jackson, Ms
Group Practice: University Clinic Associates

Data Provided by:
Kathleen R Farrell, DO
(601) 991-0066
Apt K-7 5025 Wayneland Dt
Jackson, MS
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Western U Hlt Sci Col Osteo Med Of The Pacific, Pomona Ca 91766
Graduation Year: 1983

Data Provided by:
Sanjib Das Shrestha, MD
Madison County Medical Center 1421peace Street
Ridgeland, MS
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Armed Forces Med Coll, Univ Of Pune, Pune, Maharashtra, India
Graduation Year: 1985

Data Provided by:
Karla Alford
(601) 364-1254
Jackson, MS
Practice Areas
Addictions and Dependency, Clinical Mental Health, Aging/Gerontological
Certifications
National Certified Counselor

Data Provided by:

Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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