Geriatric Healthcare Specialist Iselin NJ

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Martin David Schlakman, MD
(732) 549-2220
251 Main St
Metuchen, NJ
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Umdnj-New Jersey Med Sch, Newark Nj 07103
Graduation Year: 1992

Data Provided by:
Pranab Haldar, MD
512 New Brunswick Ave
Fords, NJ
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: St George'S Univ, Sch Of Med, St George'S, Grenada
Graduation Year: 1990

Data Provided by:
Swati G Nadkarni, MD
(908) 756-5356
92 Tingley Ln
Edison, NJ
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Female
Education
Medical School: L T M Med Coll, Univ Of Bombay, Bombay, Maharashtra, India
Graduation Year: 1991

Data Provided by:
Adrian Visoiu
(732) 442-6464
260 Hobart St
Perth Amboy, NJ
Specialty
Geriatric Medicine

Data Provided by:
Ketankumar N Vaidya
(732) 985-2151
2149 Woodbridge Ave
Edison, NJ
Specialty
Geriatric Medicine

Data Provided by:
Shailaja Shah, MD
(732) 235-6153
84 Wick Dr
Fords, NJ
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Grant Med Coll, Univ Of Bombay, Bombay, Maharashtra, India
Graduation Year: 1989

Data Provided by:
Pranab Kumar Haldar
(732) 738-5401
512 New Brunswick Ave
Fords, NJ
Specialty
Geriatric Medicine

Data Provided by:
Pradip Shah, MD
(732) 662-9104
423 Hidden Valley Dr
Edison, NJ
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Bj Med Coll, Gujarat Univ, Ahmedabad, Gujarat, India
Graduation Year: 1982

Data Provided by:
Adrian Visoiu, MD
(732) 442-6464
260 Hobart St
Perth Amboy, NJ
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Inst De Med Si Farm, Carol Davila, Bucharest, Romania
Graduation Year: 1993

Data Provided by:
Ketankumar Vaidya, MD
2149 Woodbridge Ave
Edison, NJ
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Pramukh Swami Med Coll, Sardar Patel Univ, Karamsad, Gujarat, India
Graduation Year: 1995

Data Provided by:
Data Provided by:

Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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