Geriatric Healthcare Specialist Indianapolis IN

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Usha Subramanian, MD
(317) 554-0000
1481 West 10th Street Hsrd 11-H
Indianapolis, IN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Gandhi Med Coll, Univ Hlth Sci, Vijayawada, Hyderabad, Ap, India
Graduation Year: 1988

Data Provided by:
Robert F LeBow
(317) 262-0950
907 E Michigan St
Indianapolis, IN
Specialty
Geriatric Medicine

Data Provided by:
Alejandro Arizmendi, MD
(513) 872-3541
1001 West 10th Street Opw-M200
Indianapolis, IN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Auto De Nuevo Leon, Fac De Med, Monterrey, Nuevo Leon, Mexico
Graduation Year: 1995

Data Provided by:
Ella Harvey Bowman
(317) 630-8636
1001 W 10th St
Indianapolis, IN
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Douglas K Miller
(317) 639-6671
1001 W 10th St
Indianapolis, IN
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Malaz Ahmed Boustani, MD
(317) 630-8000
1050 Wishard Blvd Rd 4th Fl
Indianapolis, IN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Damascus, Fac Of Med, Damascus, Syria
Graduation Year: 1994

Data Provided by:
Jeanne Isabelle Dickens, MD
(317) 274-4333
1231 W Michigan St
Indianapolis, IN
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Univ Of Nc At Chapel Hill Sch Of Med, Chapel Hill Nc 27599
Graduation Year: 1990

Data Provided by:
Robin Ann Beck, MD
(317) 630-8000
1001 W 10th St Fl RG4
Indianapolis, IN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: In Univ Sch Of Med, Indianapolis In 46202
Graduation Year: 1995

Data Provided by:
John Eugene Lammers, MD
(317) 962-2929
1633 N Capitol Ave Ste 322
Indianapolis, IN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of South Al Coll Of Med, Mobile Al 36688
Graduation Year: 1982
Hospital
Hospital: Methodist Hosp Of Indiana, Indianapolis, In
Group Practice: Center For Geriatric Medicine

Data Provided by:
Sanjay Gupta, MD
(317) 274-3774
702 Barnhill Dr Rm ROC4210
Indianapolis, IN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Gov'T Med Coll, Kashmir Univ, Srinagar, Jammu & Kashmir, India
Graduation Year: 1987

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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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