Geriatric Healthcare Specialist Huntsville AL

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Anupama Dronavalli Yedla, MD
301 Governors Dr SE
Huntsville, AL
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Dr Br Ambedkar Med Coll, Bangalore Univ, Bangalore, Karnataka
Graduation Year: 1994

Data Provided by:
Wayde Zane Slocumb
(256) 797-6095
Huntsville, AL
Practice Areas
Childhood & Adolescence, Aging/Gerontological, Couples & Family, Depression/Grief/Chronically or Terminally Ill
Certifications
National Certified Counselor

Igor Bidikov
(256) 463-7454
40 Giles St
Heflin, AL
Specialty
Geriatric Medicine

Data Provided by:
David Evans Urias Morris, MD
(334) 284-3211
Tuskegee, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of West Indies, Fac Med Sci, Kingston, Jamaica (566-01 Eff 1/71)
Graduation Year: 1962

Data Provided by:
Thomas Earl Stevens, MD
(251) 660-5858
307 North Univ Blvd Ste 1100,
Mobile, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Ms Sch Of Med, Jackson Ms 39216
Graduation Year: 1988

Data Provided by:
Laura Elaine Dyer, MD
(256) 536-5635
4845 Cove Creek Dr SE
Brownsboro, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Al Sch Of Med, Birmingham Al 35294
Graduation Year: 1998

Data Provided by:
Lisa Philippart
(256) 665-3607
Huntsville, AL
Practice Areas
Eating Disorders, Aging/Gerontological, Depression/Grief/Chronically or Terminally Ill, Mental Health/Agency Counseling
Certifications
National Certified Counselor

Robert Charles Blackmon, MD
(251) 432-4117
510 S Wilson Ave
Prichard, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Howard Univ Coll Of Med, Washington Dc 20059
Graduation Year: 1997

Data Provided by:
Mary Margaret O'Donoghue, MD
3146 Pine Ridge Rd
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Coll Of Galway, Nat'L Univ Of Ireland, Fac Of Med, Galway
Graduation Year: 1980

Data Provided by:
Sara Rae Rahman, MD
1521 11th Ave S,
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Med Coll Of Wi, Milwaukee Wi 53226
Graduation Year: 2000

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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