Geriatric Healthcare Specialist Howell MI

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

William A Bush
(517) 548-1246
820 Byron Rd
Howell, MI
Specialty
Geriatric Medicine

Data Provided by:
Nasir Ali
(810) 225-0086
1021 Karl Greimel Dr Ste 102
Brighton, MI
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Alan Nahit Dengiz, MD
(734) 712-5189
5361 Mcauley Drive
Ann Arbor, MI
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Mi Med Sch, Ann Arbor Mi 48109
Graduation Year: 1974

Data Provided by:
Khaled Imam, MD
(248) 551-0622
3535 W 13 Mile Rd # 108
Royal Oak, MI
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine, Endocrinology, Diabetes & Metabolism
Gender
Male
Education
Medical School: Univ Of Damascus, Fac Of Med, Damascus, Syria
Graduation Year: 1978
Hospital
Hospital: William Beaumont Hospital -Ro, Royal Oak, Mi; William Beaumont Hosp/Troy, Troy, Mi

Data Provided by:
Karen Whitney Merritt, MD
(313) 341-2451
15101 Ford Rd
Dearborn, MI
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Wayne State Univ Sch Of Med, Detroit Mi 48201
Graduation Year: 1988

Data Provided by:
Mohammad Nadeemullah, MD
(810) 844-7500
7575 West Grandrwer South
Brighton, MI
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Dow Med Coll, Univ Of Karachi, Karachi, Pakistan
Graduation Year: 1983
Hospital
Hospital: St Joseph Mercy Livingston Hos, Howell, Mi; Select Specialty Hosp Of Ann A, Ypsilanti, Mi
Group Practice: Internal Medicine Assoc

Data Provided by:
Mark Douglas Ensberg, MD
(517) 377-0330
4900 Zimmer Rd
Williamston, MI
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Sd Sch Of Med, Vermillion Sd, 57069
Graduation Year: 1981

Data Provided by:
Kenneth J Wolok, DO
(586) 979-5100
37450 Dequindre Rd
Sterling Heights, MI
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Mi State Univ, Coll Of Osteo Med, East Lansing Mi 48824
Graduation Year: 1977

Data Provided by:
Randall Mark Christenson, MD
(616) 281-6345
7819 Kenrob Dr SE
Grand Rapids, MI
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Creighton Univ Sch Of Med, Omaha Ne 68178
Graduation Year: 1978

Data Provided by:
Nadeem Mohammad Mirza, MD
(269) 998-8785
505 Hazen St
Paw Paw, MI
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Allama Iqbal Med Coll, Univ Of Punjab, Lahore, Pakistan
Graduation Year: 1985

Data Provided by:
Data Provided by:

Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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