Geriatric Healthcare Specialist Hopkinsville KY

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

John E Cotthoff, MD
(502) 886-6900
PO Box 608
Hopkinsville, KY
Specialties
Internal Medicine, Geriatrics
Gender
Male
Education
Medical School: Univ Of Tn, Memphis, Coll Of Med, Memphi
Graduation Year: 1948

Data Provided by:
Syed Sajid Ali Shah, MD
Jackson, KY
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Rawalpindi Med Coll, Univ Of Punjab, Rawalpindi, Pakistan
Graduation Year: 1994

Data Provided by:
Muhammad I Masroor, MD
(865) 544-9351
Louisville, KY
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Dow Med Coll, Univ Of Karachi, Karachi, Pakistan
Graduation Year: 1996

Data Provided by:
Ralph E Compton
(859) 986-2343
305 Estill St
Berea, KY
Specialty
Geriatric Medicine

Data Provided by:
Renukamba Gubbi, MD
(606) 849-8709
1907 Hamer St Apt 17
Flatwoods, KY
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Mysore Med Coll, Mysore Univ, Mysore, Karnataka, India
Graduation Year: 1993

Data Provided by:
Elizabeth A Toms
(270) 885-2091
1724 Kenton St
Hopkinsville, KY
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Kenneth E Holtzapple, MD
(502) 588-5201
6610 Foxcroft Rd
Prospect, KY
Specialties
Internal Medicine, Geriatrics
Gender
Male
Education
Medical School: Temple Univ Sch Of Med, Philadelphia Pa
Graduation Year: 1959

Data Provided by:
Abigail V Rayner
(502) 969-6552
200 High Rise Dr
Louisville, KY
Specialty
Family Practice, Geriatric Medicine

Data Provided by:
Svitlana J Mandzy
(502) 895-4263
4003 Kresge Way
Louisville, KY
Specialty
Geriatric Medicine

Data Provided by:
Abhilasha M N Singh, MD
(270) 265-5010
123 Daleview Cir
Russellville, KY
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Krishna Inst Of Med Sci, Shivaji Univ, Karad, Maharashtra, India
Graduation Year: 1995

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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