Geriatric Healthcare Specialist Hibbing MN

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Benjamin P Owens, MD
(218) 263-7583
Mesabi Clin 14th Avenue East And 18th
Hibbing, MN
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Univ Of Mn Med Sch-Minneapolis, Minneapolis Mn 55455
Graduation Year: 1949
Hospital
Hospital: University Med Ctr -Mesabi, Hibbing, Mn
Group Practice: Mesaba Clinic

Data Provided by:
Roberta M Meyers
(612) 873-2723
701 Park Ave
Minneapolis, MN
Specialty
Geriatric Medicine

Data Provided by:
Michael T Spilane
(651) 254-7600
401 Phalen Boulevard
St Paul, MN
Specialty
Geriatric Medicine

Data Provided by:
Debra Ann Zimmer, MD
(612) 725-2035
1478 Almond Ave
Saint Paul, MN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Mn Med Sch-Minneapolis, Minneapolis Mn 55455
Graduation Year: 1986

Data Provided by:
Muna Jneidi, MD
200 1st St SW
Rochester, MN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Tichreen, Fac Of Med, Lattakia, Syria (Univ Latakia)
Graduation Year: 1995

Data Provided by:
Rahim S Hosseini Dehkordi
(952) 993-3025
3850 Park Nicollet Blvd
St Louis Park, MN
Specialty
Family Practice, Geriatric Medicine

Data Provided by:
Melissa P Preiner Flint, MD
(612) 798-8800
407 W 66th St
Minneapolis, MN
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Female
Education
Medical School: Univ Of Mn Med Sch-Minneapolis, Minneapolis Mn 55455
Graduation Year: 1995

Data Provided by:
James W Adams
(651) 773-0450
1560 Beam Avenue
Maplewood, MN
Specialty
Geriatric Medicine

Data Provided by:
Carlos E Figari, MD
(612) 728-7700
2700 E Lake St Ste 1100
Minneapolis, MN
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Univ Nac Federico Villarreal, Prog Acad De Med Humana, Lima, Peru
Graduation Year: 1990

Data Provided by:
Thomas L Von Sternberg, MD
(612) 371-1600
8100 34th Ave S
Minneapolis, MN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Oh State Univ Coll Of Med, Columbus Oh 43210
Graduation Year: 1980

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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