Geriatric Healthcare Specialist Hibbing MN

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Benjamin P Owens, MD
(218) 263-7583
Mesabi Clin 14th Avenue East And 18th
Hibbing, MN
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Univ Of Mn Med Sch-Minneapolis, Minneapolis Mn 55455
Graduation Year: 1949
Hospital
Hospital: University Med Ctr -Mesabi, Hibbing, Mn
Group Practice: Mesaba Clinic

Data Provided by:
Wesley Earl Brugger, MD
(612) 863-4096
2800 Chicago Ave
Minneapolis, MN
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Univ Of Mn Med Sch-Minneapolis, Minneapolis Mn 55455
Graduation Year: 1970

Data Provided by:
Mohammad Albaba
(507) 284-2511
200 1st St Sw
Rochester, MN
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Pankaj Gupta, MD
(612) 467-4135
1 Veterans Dr
Minneapolis, MN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: St George'S Univ, Sch Of Med, St George'S, Grenada
Graduation Year: 1996

Data Provided by:
Brian Deane Nelson, MD
(320) 763-5123
4021 Rue de Frenchman NE
Alexandria, MN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Chicago, Pritzker Sch Of Med, Chicago Il 60637
Graduation Year: 1996

Data Provided by:
Thomas L Von Sternberg, MD
(612) 371-1600
8100 34th Ave S
Minneapolis, MN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Oh State Univ Coll Of Med, Columbus Oh 43210
Graduation Year: 1980

Data Provided by:
Jack B Petrie, MD
(612) 641-7043
15100 Square Lake Trl N
Stillwater, MN
Specialties
Internal Medicine, Geriatrics
Gender
Male
Education
Medical School: Rush Med Coll Of Rush Univ, Chicago Il 6
Graduation Year: 1981

Data Provided by:
Daniel J Stein
(612) 455-2040
701 25th Ave S
Minneapolis, MN
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
George Nicholas Battis, MD
(612) 298-3990
401 Robert St N
Saint Paul, MN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Mn Med Sch-Minneapolis, Minneapolis Mn 55455
Graduation Year: 1978

Data Provided by:
Benjamin P Owens, MD
(218) 263-7583
Mesabi Clin 14th Avenue East And 18th
Hibbing, MN
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Univ Of Mn Med Sch-Minneapolis, Minneapolis Mn 55455
Graduation Year: 1949
Hospital
Hospital: University Med Ctr -Mesabi, Hibbing, Mn
Group Practice: Mesaba Clinic

Data Provided by:
Data Provided by:

Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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