Geriatric Healthcare Specialist Glen Allen VA

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Aparna Ranjan
(804) 627-5291
8580 Magellan Pkwy
Richmond, VA
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Nalini Morishetty, MD
417 N 11th St
Richmond, VA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Siddartha Med Coll, Univ Of Hlth Sci, Vijayawada, Ap, India
Graduation Year: 1997

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Amy Marie Irwin, MD
1250 E Marshall St
Richmond, VA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Ok Coll Of Med, Oklahoma City Ok 73190
Graduation Year: 2000

Data Provided by:
Sheldon M Retchin
(828) 530-6804
1250 E Marshall St
Richmond, VA
Specialty
Internal Medicine, Geriatric Medicine

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Catherine Mcvearry Kelso
(828) 532-3804
1250 E Marshall St
Richmond, VA
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Rachel M Selby Penczak, MD
401 N 11th St
Richmond, VA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Med Coll Of Pa, Philadelphia Pa 19129
Graduation Year: 1995

Data Provided by:
Christine Linda Tully, MD
401 N 12th St
Richmond, VA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Di Roma-La Sapienza, Fac Di Med E Chirurgia, Roma, Italy
Graduation Year: 1978

Data Provided by:
Swarna B Reddy, MD
(804) 828-9723
PO Box 9802301101e Marshall St
Richmond, VA
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Sri Venkatesvara Med Coll, Univ Hlth Sci, Tirupati, Ap, India
Graduation Year: 1991

Data Provided by:
Albert Mingtat Leung, MD
15 N 18th St Apt 2G
Richmond, VA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Hi John A Burns Sch Of Med, Honolulu Hi 96822
Graduation Year: 1992

Data Provided by:
Kalu Awa Uma, MD
(804) 675-5000
1200 E Marshall St
Richmond, VA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Nigeria, Coll Of Med, Enugu, Anambra, Nigeria
Graduation Year: 1993

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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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