Geriatric Healthcare Specialist Freeport IL

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Abimbola Awodipe
1301 Kiwanis Dr
Freeport, IL
Specialty
Geriatric Internal Medicine, Alzheimer's Specialist

Khaja Asadullah, MD
(773) 257-6542
30 E 15th St
Chicago Heights, IL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Deccan Coll Of Med Sci, Osmania Univ, Hyderabad, Ap, India
Graduation Year: 1992

Data Provided by:
Aaron Lazar
(630) 717-2646
100 Spalding Dr
Naperville, IL
Specialty
Geriatric Medicine

Data Provided by:
Harish Bhatia, MD
(708) 535-3300
6300 159th St Ste C
Oak Forest, IL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine, Gastroenterology
Gender
Male
Languages
Hindi, Spanish
Education
Medical School: Maulana Azad Med Coll, Univ Of Delhi, New Delhi, Delhi, India
Graduation Year: 1978
Hospital
Hospital: Ingalls Mem Hosp, Harvey, Il; Palos Comm Hosp, Palos Heights, Il

Data Provided by:
Umar Waheed, MD
(708) 346-5673
4440 W 95th St Ste 3195
Oak Lawn, IL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Rawalpindi Med Coll, Univ Of Punjab, Rawalpindi, Pakistan
Graduation Year: 1995

Data Provided by:
Oscar Francis Florendo
(618) 651-8333
180 Woodcrest Dr
Highland, IL
Specialty
Geriatric Medicine

Data Provided by:
Jobinson Thomas, MD
(312) 996-6584
1855 W Taylor St
Chicago, IL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Christian Med Coll, Punjab Univ, Ludhiana, Punjab, India
Graduation Year: 1990

Data Provided by:
Victoria L Braund
(847) 998-4100
2050 Pfingsten Rd
Glenview, IL
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Mona Tareen
(312) 942-7030
1725 W Harrison St
Chicago, IL
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Gladstone A Tucker, MD
(816) 373-4415
1607 Castleberry Dr
Marion, IL
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Patrice Lumumba People'S Friendship Univ, Med Fak, Moskva, Russia
Graduation Year: 1980

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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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