Geriatric Healthcare Specialist Forest Park GA

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Shashikant A Daya, MD
3305 Bobby Brown Pkwy
East Point, GA
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Univ Of The Witwatersrand, Med Sch, Johannesburg, So Africa
Graduation Year: 1986

Data Provided by:
Abimbola Adetola Adesina, MD
720 Westview Dr SW
Atlanta, GA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Lagos, Coll Of Med, Lagos, Nigeria
Graduation Year: 1989

Data Provided by:
Abimbola Akomolafe-Adesina, MBBS
(404) 756-1320
720 Westview Dr SW
Atlanta, GA
Specialties
Internal Medicine, Geriatrics
Gender
Male
Education
Medical School: Univ Of Lagos, Coll Of Med, Lagos, Niger
Graduation Year: 1989

Data Provided by:
Abraham Ayotunde Oyewo, MD
75 Piedmont Avenue North East South
Atlanta, GA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Ahmadu Bello Univ, Fac Of Med, Zaria, Kaduna, Nigeria
Graduation Year: 1989

Data Provided by:
Lavonda Mee Lee, MD
Decatur, GA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Hi John A Burns Sch Of Med, Honolulu Hi 96822
Graduation Year: 1999

Data Provided by:
Cecile Q Nguyen
(404) 766-4633
1029 Cleveland Ave
East Point, GA
Specialty
Geriatric Medicine

Data Provided by:
Meena Gulati, MD
(404) 752-1500
720 Westview Dr SW
Atlanta, GA
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Lady Hardinge Med Coll, Univ Of Delhi, New Delhi, Delhi, India
Graduation Year: 1981

Data Provided by:
Kieran Alexander Cooper
(678) 499-8633
41 Marietta St Nw
Atlanta, GA
Specialty
Geriatric Medicine

Data Provided by:
Kaysar Mamun, MD
Decatur, GA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Sir Salimullah Med Coll, Dhaka, Bangladesh
Graduation Year: 1990

Data Provided by:
Louise Foushee Horney, MD
(404) 728-6363
744 Courtenay Ct NE
Atlanta, GA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Nc At Chapel Hill Sch Of Med, Chapel Hill Nc 27599
Graduation Year: 1990

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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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