Geriatric Healthcare Specialist Foley AL

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Sayed Asad Ali, MD
(317) 247-1885
1700 Center St
Mobile, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: King Edward Med Coll, Univ Of Punjab, Lahore, Pakistan
Graduation Year: 1996

Data Provided by:
Thomas Earl Stevens, MD
(251) 660-5858
307 North Univ Blvd Ste 1100,
Mobile, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Ms Sch Of Med, Jackson Ms 39216
Graduation Year: 1988

Data Provided by:
Joel Charles Bolen, MD
(334) 277-7665
3150 Knightsbridge Curv
Montgomery, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Bowman Gray Sch Of Med Of Wake Forest Univ, Winston-Salem Nc 27157
Graduation Year: 1995

Data Provided by:
Robert Charles Blackmon, MD
(251) 432-4117
510 S Wilson Ave
Prichard, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Howard Univ Coll Of Med, Washington Dc 20059
Graduation Year: 1997

Data Provided by:
Sara Rae Rahman, MD
1521 11th Ave S,
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Med Coll Of Wi, Milwaukee Wi 53226
Graduation Year: 2000

Data Provided by:
Patricia Flora Harris, MD
(202) 877-0218
2000 6th Ave S
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Wi Med Sch, Madison Wi 53706
Graduation Year: 1995
Hospital
Hospital: Washington Hosp Ctr, Washington, Dc
Group Practice: Washington Hospital Ctr

Data Provided by:
Cynthia Holcomb Baker, MD
619 19th St S
Birmingham, AL
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of South Al Coll Of Med, Mobile Al 36688
Graduation Year: 1996

Data Provided by:
Terri Sue Steele, MD
(205) 934-6054
1713 6th Ave S
Birmingham, AL
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Univ Of South Al Coll Of Med, Mobile Al 36688
Graduation Year: 1986

Data Provided by:
Johnnie Wayne Stevens, MD
(205) 979-3362
115 Paradise Isle
Riverside, AL
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Univ Of Al Sch Of Med, Birmingham Al 35294
Graduation Year: 1970
Hospital
Hospital: St Vincents Hosp, Birmingham, Al; Brookwood Med Ctr, Birmingham, Al

Data Provided by:
Russell Dean Ulrich
(256) 447-9045
800 Memorial Dr
Piedmont, AL
Specialty
Geriatric Medicine

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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