Geriatric Healthcare Specialist East Liverpool OH

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Shantanu Saraswat, MD
(330) 386-4215
15122 Sprucevale Rd Apt A
East Liverpool, OH
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Maulana Azad Med Coll, Univ Of Delhi, New Delhi, Delhi, India
Graduation Year: 1997

Data Provided by:
Mark Howard Overton, MD
(724) 378-6565
Monaca, PA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Or Hlth Sci Univ Sch Of Med, Portland Or 97201
Graduation Year: 1981

Data Provided by:
Laurence Jay Robbins, MD
(937) 643-9299
3533 Southern Blvd Ste 5350
Kettering, OH
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Rochester Sch Of Med & Dentistry, Rochester Ny 14642
Graduation Year: 1975

Data Provided by:
Ammaji Narra
(216) 621-5600
14600 Detroit Ave
Lakewood, OH
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Jyothi Dyavanapalli Gudla
(330) 489-4600
733 Market Ave S
Canton, OH
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Nevedita Saigal, MD
16687 Saint Clair Ave
East Liverpool, OH
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Maulana Azad Med Coll, Univ Of Delhi, New Delhi, Delhi, India
Graduation Year: 1997

Data Provided by:
Chantal Germaine Walsh, MD
400 Wabash Ave
Akron, OH
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Female
Education
Medical School: St Louis Univ Sch Of Med, St Louis Mo 63104
Graduation Year: 1996

Data Provided by:
Judith Kidd Held, MD
(740) 363-1531
695 W Central Ave
Delaware, OH
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Oh State Univ Coll Of Med, Columbus Oh 43210
Graduation Year: 1966

Data Provided by:
Matthew Stevenson Wayne
(216) 844-6300
12200 Fairhill Rd
Cleveland, OH
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Aly M Zewail
(330) 345-5374
128 E Milltown Rd
Wooster, OH
Specialty
Family Practice, Geriatric Medicine

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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