Geriatric Healthcare Specialist Deridder LA

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Susan Ellen Nelson
(225) 765-3076
5339 Didesse Dr
Baton Rouge, LA
Specialty
Internal Medicine, Geriatric Medicine

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Joseph Sampognaro, MD
(504) 282-5398
145 Robert E Lee Blvd
New Orleans, LA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: La State Univ Sch Of Med In New Orleans, New Orleans La 70112
Graduation Year: 1975

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Santosh K Gopalakrishnan, MD
(803) 531-6956
1601 Perdido St Dept GERI
New Orleans, LA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Bangalore Med Coll, Bangalore Univ, Bangalore, Karnataka, India
Graduation Year: 1994

Data Provided by:
Kundan Lal Manocha, MD
510 E Stoner Ave
Shreveport, LA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Med Coll, Guru Nanak Dev Univ, Amritsar, Punjab, India
Graduation Year: 1965

Data Provided by:
Lawrence J Drexler
(318) 222-8187
850 Margaret Pl
Shreveport, LA
Specialty
Internal Medicine, Geriatric Medicine

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Henry Rothschild
(504) 412-1693
2020 Gravier St
New Orleans, LA
Specialty
Geriatric Medicine

Data Provided by:
Veronica Ebihomo Adoun
(337) 261-0734
2100 Jefferson St
Lafayette, LA
Specialty
Geriatric Medicine

Data Provided by:
William Patrick Gahan
(225) 765-3076
5339 Didesse Dr
Baton Rouge, LA
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Murali Srinivasan
(318) 222-8187
850 Margaret Pl
Shreveport, LA
Specialty
Geriatric Medicine

Data Provided by:
James Ford Riopelle, MD
(303) 781-6430
9032 Perkins Rd
Baton Rouge, LA
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Mi State Univ Coll Of Human Med, East Lansing Mi 48824
Graduation Year: 1973

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Food for Thought

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By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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