Geriatric Healthcare Specialist Connersville IN

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Anthony Joseph Perry, MD
(219) 486-9102
4222 Stillwood Dr
Fort Wayne, IN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Jefferson Med Coll-Thos Jefferson Univ, Philadelphia Pa 19107
Graduation Year: 1991

Data Provided by:
Syed Fawad Hussain, MD
3900 Southland Ave
Kokomo, IN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Dow Med Coll, Univ Of Karachi, Karachi, Pakistan
Graduation Year: 1991

Data Provided by:
Lynn Griffey Valena, MD
(765) 288-2707
10681 W Sweet Gum St
Yorktown, IN
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Female
Education
Medical School: In Univ Sch Of Med, Indianapolis In 46202
Graduation Year: 1980

Data Provided by:
Ahmed Raza Ibrahim
(317) 338-7780
8240 Naab Rd
Indianapolis, IN
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Joseph Francis Jr, MD
(317) 338-7017
Indianapolis, IN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Washington Univ Sch Of Med, St Louis Mo 63110
Graduation Year: 1984

Data Provided by:
Scott Casper Bruins, MD
(317) 262-0950
4747 N Meridian St
Indianapolis, IN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Johns Hopkins Univ Sch Of Med, Baltimore Md 21205
Graduation Year: 1968

Data Provided by:
John S Kelly
(219) 785-2407
156 Flynn Road
Westville, IN
Specialty
Family Practice, Geriatric Medicine

Data Provided by:
Harry Stoller
(574) 291-6754
18444 Madison Rd
South Bend, IN
Specialty
Geriatric Medicine

Data Provided by:
Raymond William Nicholson, MD
(812) 485-4173
3900 Washington Ave Ste 200
Evansville, IN
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: In Univ Sch Of Med, Indianapolis In 46202
Graduation Year: 1955
Hospital
Hospital: St Marys Med Ctr Of Evansville, Evansville, In; St Elizabeth Hosp Med Center, Lafayette, In
Group Practice: Family Practice Center

Data Provided by:
Alejandro Arizmendi, MD
(513) 872-3541
1001 West 10th Street Opw-M200
Indianapolis, IN
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Auto De Nuevo Leon, Fac De Med, Monterrey, Nuevo Leon, Mexico
Graduation Year: 1995

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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