Geriatric Healthcare Specialist Claremore OK

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

James Kyle Cooper, MD
(918) 488-0301
7706 S Harvard Pl
Tulsa, OK
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Vanderbilt Univ Sch Of Med, Nashville Tn 37232
Graduation Year: 1962

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Banu Sezginsoy
(405) 271-3050
825 Ne 10th St
Oklahoma City, OK
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Christos DiMas
(405) 685-6260
2716 Sw 44th
Oklahoma City, OK
Specialty
Geriatric Medicine

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Muhammad Firdaus, MD
(405) 271-8558
14916 Salem Creek Rd
Edmond, OK
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Dow Med Coll, Univ Of Karachi, Karachi, Pakistan
Graduation Year: 1994

Data Provided by:
Angelique Barreto, MD
(405) 272-7000
3705 W Memorial Rd
Oklahoma City, OK
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Madras Med Coll, Dr M G R Med Univ, Madras, Tn, India
Graduation Year: 1991
Hospital
Hospital: St Anthony Hospital, Oklahoma City, Ok
Group Practice: Saint Anthony Hospital

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William G Berlinger III, MD
(918) 524-3563
Tulsa, OK
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Pa Sch Of Med, Philadelphia Pa 19104
Graduation Year: 1979

Data Provided by:
Ishita G Thakar, MD
(405) 943-7592
Department Geri 921 North East 13th Avenue
Oklahoma City, OK
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Maulana Azad Med Coll, Univ Of Delhi, New Delhi, Delhi, India
Graduation Year: 1995

Data Provided by:
Karin Ann Johnson, DO
(405) 271-4351
221 NW 160th Ter
Edmond, OK
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Ok State Univ, Coll Of Osteo Med, Tulsa, Ok 74107
Graduation Year: 1998

Data Provided by:
Martina Jan Jelley
(918) 619-4101
4444 E. 41st St
Tulsa, OK
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Banu Sezginsoy, MD
825 Ne 10 Oupb4300
Oklahoma City, OK
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Cukurova Univ, Tip Fak, Adana, Turkey
Graduation Year: 1993

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Food for Thought

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By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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