Geriatric Healthcare Specialist Calexico CA

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Xian Li
(818) 375-2000
13652 Cantara St
Panorama City, CA
Specialty
Geriatric Medicine

Data Provided by:
Harry Minoru Shin, MD
(909) 427-5441
3666 El Grande Dr
San Jose, CA
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Loma Linda Univ Sch Of Med, Loma Linda Ca 92350
Graduation Year: 1992

Data Provided by:
Dave Robinson, DO
(209) 951-4666
1145 N California St
Stockton, CA
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Univ Of Hlth Sci, Coll Of Osteo Med, Kansas City Mo 64124
Graduation Year: 1990

Data Provided by:
David Naimark, MD
(619) 531-3257
1924 Montgomery Ave
Cardiff, CA
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Male
Education
Medical School: Boston Univ Sch Of Med, Boston Ma 02118
Graduation Year: 1990

Data Provided by:
Harold Brownstein, MD
(415) 461-1920
Greenbrae, CA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Stanford Univ Sch Of Med, Stanford Ca 94305
Graduation Year: 1955

Data Provided by:
Laurence Z Rubenstein, MD
(818) 895-9311
16111 Plummer St Bldg 25RM # A129
Sepulveda, CA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: A Einstein Coll Of Med Of Yeshiva Univ, Bronx Ny 10461
Graduation Year: 1974

Data Provided by:
James Edward Lett, MD
(916) 978-0958
5098 Keane Dr
Carmichael, CA
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Univ Of Ky Coll Of Med, Lexington Ky 40536
Graduation Year: 1974

Data Provided by:
Susan Lu, MD
(510) 465-6700
350 30th St Ste 320
Oakland, CA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Wayne State Univ Sch Of Med, Detroit Mi 48201
Graduation Year: 1998

Data Provided by:
Jeffrey Paul Frey, MD
(573) 442-0320
376 W Badillo St
Covina, CA
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine, Adolescent Medicine-Pediatrics
Gender
Male
Education
Medical School: Suny-Hlth Sci Ctr At Syracuse, Coll Of Med, Syracuse Ny 13210
Graduation Year: 1974
Hospital
Hospital: Boone Hosp Center, Columbia, Mo; Columbia Reg Hosp, Columbia, Mo; University Hospitals And Clini, Columbia, Mo
Group Practice: Broadway Internal Medicine

Data Provided by:
Glenn C Kissinger
(559) 591-1820
271 North L Street
Dinuba, CA
Specialty
Geriatric Medicine

Data Provided by:
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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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