Geriatric Healthcare Specialist Brunswick GA
Childhood & Adolescence, Aging/Gerontological, Couples & Family, Depression/Grief/Chronically or Terminally Ill, Supervision
National Certified Counselor
Geriatrics, Geriatric Medicine-Internal Medicine
Medical School: Goa Med Coll, Goa Univ, Panaji, Daman & Diu, Goa, India
Graduation Year: 1993
Internal Medicine, Geriatric Medicine
Family Practice, Geriatric Medicine
Geriatrics, Geriatric Psychiatry
Medical School: George Washington Univ Sch Of Med & Hlth Sci, Washington Dc 20037
Graduation Year: 1987
Hospital: Wesley Woods Geriatric Hosp, Atlanta, Ga; Emory University Hosp, Atlanta, Ga; Crawford Long Hosp, Atlanta, Ga
Geriatrics, Geriatric Psychiatry
Medical School: Univ Of Ar Coll Of Med, Little Rock Ar 72205
Graduation Year: 1984
Hospital: Wesley Woods Geriatric Hosp, Atlanta, Ga; Emory University Hosp, Atlanta, Ga
Group Practice: Emory Clinic
Warner Robins, GA
Family Practice, Internal Medicine, Geriatric Medicine, Emergency Medicine
Internal Medicine, Geriatrics
Medical School: Tulane Univ Sch Of Med, New Orleans La 7
Graduation Year: 1950
Geriatric Medicine, Emergency Medicine
Food for Thought
By Kris Kucera
Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.
In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.
Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.
Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.
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