Geriatric Healthcare Specialist Brigham City UT

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Gary Griffin
(801) 268-7766
1160 E 3900 S
Salt Lake City, UT
Specialty
Geriatric Medicine

Data Provided by:
John Emil Carlson, MD
50 North Medical Dr
Salt Lake City, UT
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Mn Med Sch-Minneapolis, Minneapolis Mn 55455
Graduation Year: 1983

Data Provided by:
Carl E Wyne, MD
(801) 314-4544
5801 Fashion Blvd Ste 175
Salt Lake City, UT
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Il Coll Of Med, Chicago Il 60680
Graduation Year: 1973

Data Provided by:
Cherie Pratt Brunker
(801) 408-8600
400 C St
Salt Lake City, UT
Specialty
Geriatric Medicine

Data Provided by:
Dr. Marie Green
(801) 476-8885
5582 South 1750 East
Ogden, UT
Services
Yeast Syndrome, Wellness Training, Weight Management, Substance Abuse, Stress Management, Psychotherapy, Preventive Medicine, Pain Management, Nutrition, Neurofeedback, Mind/Body Medicine, Medical Intuition, Hypnosis/Hypnotherapy, Homeopathy, Healthy Aging, Guided Imagery, Geriatrics, EFT, EMDR, Cognitive Therapy, Coaching, Breathwork, Brain Longevity, Biofeedback, Bach Flower Essences, Aromatherapy, Addiction
Membership Organizations
American Holistic Medical Association

Data Provided by:
Charles Steven Fehlauer
(801) 272-0255
2773 Etienne Way
Sandy, UT
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Sandeep Chowdhary, MD
3725 West 4100 South uth
Salt Lake City, UT
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Gov'T Med Coll, Kashmir Univ, Srinagar, Jammu & Kashmir, India
Graduation Year: 1995

Data Provided by:
Nancy Jo Zega
(801) 479-2911
425 E 5350 S
Ogden, UT
Specialty
Geriatric Medicine

Data Provided by:
Danesh Rahimi
(801) 268-7766
1160 E 3900 S
Salt Lake City, UT
Specialty
Geriatric Medicine, Emergency Medicine

Data Provided by:
Frederick Lewis Gottlieb, MD
(801) 350-4602
4727 Naniloa Dr
Salt Lake City, UT
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Wv Univ Sch Of Med, Morgantown Wv 26506
Graduation Year: 1984

Data Provided by:
Data Provided by:

Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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