Geriatric Healthcare Specialist Bluefield WV

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Maria Karina F Abad, MD
(304) 927-1495
PO Box 30
Spencer, WV
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Female
Education
Medical School: Cebu Doctors Coll Of Med, Cebu City, Philippines
Graduation Year: 1991

Data Provided by:
Albert J Grant, MD
(605) 745-5783
PO Box 747
Weston, WV
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Jefferson Med Coll-Thos Jefferson Univ, Philadelphia Pa 19107
Graduation Year: 1950

Data Provided by:
William Lee Harris
(304) 344-2451
3100 Maccorkle Ave Se
Charleston, WV
Specialty
Geriatric Medicine

Data Provided by:
William C Boyd
(304) 255-2121
200 Veterans Ave
Beckley, WV
Specialty
General Practice, Family Practice, Geriatric Medicine

Data Provided by:
Vishal Bhagchand Malpani, MD
(304) 927-1495
881 Parkersburg Rd
Spencer, WV
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Topiwala Nat'L Med Coll, Univ Of Bombay, Bombay, Maharashtra, India
Graduation Year: 1996

Data Provided by:
Cynthia A Winger
(304) 736-5247
659 Central Ave
Barboursville, WV
Specialty
Family Practice, Geriatric Medicine

Data Provided by:
Ghassan Rahhal, MD
(304) 258-9248
2580 Fairview Dr Apt 1
Berkeley Springs, WV
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Aleppo, Fac Of Med, Aleppo, Syria
Graduation Year: 1994

Data Provided by:
Vincent W De La Garza, MD
(304) 598-0695
6003 Woodland Bluff Rd
Morgantown, WV
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Male
Education
Medical School: Univ Of Md Sch Of Med, Baltimore Md 21201
Graduation Year: 1976
Hospital
Hospital: W V University Hospital -Ruby, Morgantown, Wv
Group Practice: University Health Associates

Data Provided by:
Marlene A Wager
(304) 645-3220
400 N Jefferson St
Lewisburg, WV
Specialty
General Practice, Family Practice, Geriatric Medicine

Data Provided by:
Richard Stockton Trenbath
(304) 226-5725
10003 Webster Road
Camden On Gauley, WV
Specialty
Family Practice, Geriatric Medicine

Data Provided by:
Data Provided by:

Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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