Geriatric Healthcare Specialist Bend OR

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Ginger Lou Dattilo, MD
(541) 826-2111
PO Box 6779
Bend, OR
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Louisville Sch Of Med, Louisville Ky 40202
Graduation Year: 1981

Data Provided by:
Dorothy Holloway
(541) 771-3247
Bend, OR
Practice Areas
Clinical Mental Health, Aging/Gerontological, Sexual Abuse Recovery, Depression/Grief/Chronically or Terminally Ill, Mental Health/Agency Counseling
Certifications
National Certified Counselor

Kenneth L Orwick
(541) 451-7200
325 Park St
Lebanon, OR
Specialty
Family Practice, Geriatric Medicine

Data Provided by:
Ellen Schiaffino, MD
440 NW Division St
Gresham, OR
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Newcastle-Upon-Tyne, Fac Of Med (352-04 Pr 1/71)
Graduation Year: 1982

Data Provided by:
John Paul Roberts
(541) 607-0897
100 River Ave
Eugene, OR
Specialty
Geriatric Medicine

Data Provided by:
James Detwiler
(541) 548-2164
211 Nw Larch Ave
Redmond, OR
Specialty
Family Practice, Geriatric Medicine

Data Provided by:
Kenneth V Brummel Smith, MD
(503) 513-8500
10150 SE 32nd Ave
Portland, OR
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Southern Ca Sch Of Med, Los Angeles Ca 90033
Graduation Year: 1975

Data Provided by:
Danielle Lynne Kizer, MD
(503) 561-5420
1127 Oak St SE
Salem, OR
Specialties
Geriatrics, Geriatric Psychiatry
Gender
Female
Education
Medical School: Univ Of Cincinnati Coll Of Med, Cincinnati Oh 45267
Graduation Year: 1997

Data Provided by:
M Hester Fieldhouse, MD
Portland, OR
Specialties
Geriatrics, Geriatric Medicine-Family Practice
Gender
Female
Education
Medical School: Or Hlth Sci Univ Sch Of Med, Portland Or 97201
Graduation Year: 1965

Data Provided by:
Rima Huda Jallad, MD
(503) 220-8262
3710 South West Us Vets Hospital Road
Portland, OR
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Damascus, Fac Of Med, Damascus, Syria
Graduation Year: 1984

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Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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