Geriatric Healthcare Specialist Altus OK

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment.

Joe LeVerett
(580) 480-3325
201 S Park Ln
Altus, OK
Specialty
Cardiology, Geriatric Medicine

Data Provided by:
Mable Wong Tan, MD
(210) 617-5300
825 NE 10th St Ste OUPB4300
Oklahoma City, OK
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Santo Tomas, Fac Of Med And Surg, Manila, Philippines
Graduation Year: 1988

Data Provided by:
Lakshmin Ramakrishnan, MD
Norman, OK
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Kilpauk Med Coll, Dr M G R Med Univ, Madras, Tn, India
Graduation Year: 1993

Data Provided by:
Joe LeVerett
(580) 480-3325
201 S Park Ln
Altus, OK
Specialty
Cardiology, Geriatric Medicine

Data Provided by:
Karin Johnson
(918) 748-7630
1919 S Wheeling Ave
Tulsa, OK
Specialty
Internal Medicine, Geriatric Medicine

Data Provided by:
Muhammad Firdaus, MD
(405) 271-8558
14916 Salem Creek Rd
Edmond, OK
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Dow Med Coll, Univ Of Karachi, Karachi, Pakistan
Graduation Year: 1994

Data Provided by:
Muhammad Aijaz Sattar, MD
(918) 567-7000
1 Choctaw Way
Talihina, OK
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Male
Education
Medical School: Dow Med Coll, Univ Of Karachi, Karachi, Pakistan
Graduation Year: 1989

Data Provided by:
Kerry Wayne Cranmer, MD
(405) 949-2005
3545 NW 58th St Ste 750
Oklahoma City, OK
Specialties
Geriatrics, Geriatric Medicine-Family Practice, Palliative Medicine
Gender
Male
Education
Medical School: Univ Of Ok Coll Of Med, Oklahoma City Ok 73190
Graduation Year: 1975
Hospital
Hospital: Deaconess Hosp, Oklahoma City, Ok
Group Practice: Cranmer & Assoc

Data Provided by:
xenin samual, DR
(123) 223-5671
asfe ert eerg e
lucknow, OK
Specialties
Geriatrics
Gender
Male
Languages
english
Education
Graduation Year: 2006

Data Provided by:
Vicki Therese Lampley, MD
(405) 297-5957
921 NE 13th St
Oklahoma City, OK
Specialties
Geriatrics, Geriatric Medicine-Internal Medicine
Gender
Female
Education
Medical School: Univ Of Ok Coll Of Med, Oklahoma City Ok 73190
Graduation Year: 1990

Data Provided by:
Data Provided by:

Food for Thought

Provided by: 

By Kris Kucera

Rarely does an extended family get a free pass from Alzheimer’s disease or diabetes mellitus. On the surface, these two afflictions appear totally unrelated —Alzheimer’s (AD), Mother Nature’s cruel version of identity theft; and diabetes, the glucose-metabolism disorder that affects both young and old alike. However, new research indicates that the two diseases behave in a similar manner.

In a study recently published in the Journal of Alzheimer’s Disease, researchers from Brown Medical School found that reduced glucose uptake and decreased metabolism in the hippocampus—the area of the brain associated with memory—cause neurodegeneration and cognitive impairment. This, they say, suggests that a form of diabetes, tentatively dubbed type 3, leads to AD.

Type 1 diabetes results from a severe or complete lack of insulin, a hormone made in the pancreas that controls blood sugar. Type 2, dubbed adult onset diabetes (although these days it occurs in teenagers and even younger kids), also stems from a dearth of insulin, or insulin resistance —the existing insulin molecules cannot deliver glucose through the cells’ membranes. Surprisingly, the researchers found a new form of insulin, produced in the brain, and they believe that, over time, decreasing levels of this “brain insulin” and other insulin-related proteins ultimately precipitate AD. While levels of brain insulin have no known affect on a body’s overall blood sugar, scientists have long recognized that diabetes patients are more likely to develop AD than those without the disease.

Skeptics of the Brown team’s findings argue that our brains produce so little insulin in the first place, reduced levels of the hormone can’t possibly play a significant role in AD. Regardless, the new data show that AD may be a neuroendocrine disorder, thus increasing the possibility for more effective treatments. And that gives hope to all of us who may one day be touched, directly or indirectly, by the merciless hand of AD.

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