Preeclampsia Treatment Towson MD

Researchers already knew that changes in the concentrations of two proteins—placental growth factor (PlGF) and soluble fms-like tyrosine kinase 1 (sFit1)—keyed the onset of milder forms of preeclampsia but did not seem to cause the life'threatening levels of high blood pressure seen in severe cases.

Frank J Bottiglieri MD
(410) 339-7640
6569 N Charles St
Towson, MD
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Obstetrics & Gynecology

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Dr.Marwan Hajj
(410) 321-1313
6569 N Charles St # 710
Towson, MD
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Medical School: American Univ Of Beirut, Fac Of Med, Beirut
Year of Graduation: 1971
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Gynecologist (OBGYN)
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Accepting New Patients: Yes
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Shobha Jayadeva, MD
(410) 494-0057
120 Sister Pierre Dr
Towson, MD
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Medical School: Jjm Med Coll, Mysore Univ, Davangere, Karnataka, India
Graduation Year: 1976

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Elaine Que Lim
(410) 825-7000
7505 Osler Drive
Towson, MD
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Obstetrics & Gynecology

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Eugene Katz, MD
6569 N Charles St
Towson, MD
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Obstetrics & Gynecology, Reproductive Endocrinology
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Medical School: Univ De Chile, Esc De Pregrado, Fac De Med, Santiago, Chile
Graduation Year: 1978

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Robert Edgar Ottenritter, MD
(410) 296-1364
6565 N Charles St Ste 212
Towson, MD
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Medical School: Univ Auto De Guadalajara, Fac De Med, Guadalajara, Jalisco, Mexico
Graduation Year: 1982

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Tatiana V Sanses
(443) 849-2767
6569 N Charles St
Baltimore, MD
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Obstetrics & Gynecology

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Colleen M Fitzpatrick, MD
(410) 337-2877
7505 Osler Dr Ste 303
Towson, MD
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Obstetrics & Gynecology
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Female
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Medical School: Univ Of Mn Med Sch-Minneapolis, Minneapolis Mn 55455
Graduation Year: 1981

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John Gordon Frizzera
(410) 828-8077
7401 Osler Dr
Towson, MD
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Obstetrics & Gynecology

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Kirkor Kazazian, MD
(410) 825-3818
6565 N Charles St Ste 201
Towson, MD
Specialties
Obstetrics & Gynecology
Gender
Male
Education
Medical School: Univ Di Bologna, Fac Di Med E Chirurgia, Bologna, Italy
Graduation Year: 1970

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Early Warning Signs of Preeclampsia

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By Kathryn Ayers

High blood pressure, fluid retention, and excess protein in the urine all signal the onset of preeclampsia, a condition that affects one in 20 women during the third trimester of pregnancy. While a cause for concern in all cases—about 200,000 women suffer from preeclampsia in the US each year, and it is a leading cause of premature births—only those women who experience large and sudden increases in blood pressure face the threat of severe complications for themselves and their fetuses.

Researchers already knew that changes in the concentrations of two proteins—placental growth factor (PlGF) and soluble fms-like tyrosine kinase 1 (sFit1)—keyed the onset of milder forms of preeclampsia but did not seem to cause the life-threatening levels of high blood pressure seen in severe cases. Their new study in The New England Journal of Medicine names a third factor, soluble endoglin, that seems to kick preeclampsia into overdrive. This protein, which the researchers found in high concentrations in the placentas of women with severe preeclampsia, normally helps maintain the blood vessels, but when it sloughs off the blood vessel walls and combines with sFit1, it actually weakens them.

The study reports that soluble endoglin levels begin to increase markedly two to three months before the onset of preeclampsia and that this increase is usually accompanied by an increased ratio of sFit1 to PlGF. This forewarning should enable researchers to develop a diagnostic test that will allow healthcare providers to counter a major cause of maternal and fetal mortality worldwide.

Author: Kathryn Ayers

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