Preeclampsia Treatment Paradise Valley AZ

Researchers already knew that changes in the concentrations of two proteins—placental growth factor (PlGF) and soluble fms-like tyrosine kinase 1 (sFit1)—keyed the onset of milder forms of preeclampsia but did not seem to cause the life'threatening levels of high blood pressure seen in severe cases.

Armity Simon, MD
(480) 860-2322
9070 E Desert Cove
Scottsdale, AZ
Business
Armity Simon, MD, PC
Specialties
Obstetrics & Gynecology
Insurance
Insurance Plans Accepted: Aetna, Blue Cross Blue Shield, Medicare, APIPA, Great West, United Health Care, Maricopa Foundation
Medicare Accepted: Yes
Accepts Uninsured Patients: Yes
Emergency Care: Yes

Doctor Information
Primary Hospital: Scottsdale Healthcare Shea
Residency Training: St. Joseph Mercy Hospital
Medical School: Indiana University School of Medicine, 1988
Additional Information
Member Organizations: American College of Obstetrics & Gynecology American Medical Association Member American Fertility Society Member Society of Laparoscopic Surgeons Member
Awards: Dean's Award-Indiana University School of Medicine President of Graduating Class Indiana School of Medicine Summa Cum Laude Phi Kappa Phi Honor Society-Indiana State University
Languages Spoken: English,Spanish,Persian

Data Provided by:
William T Cook, MD
(404) 355-1285
4223 E Highlands Dr
Paradise Valley, AZ
Specialties
Obstetrics & Gynecology
Gender
Male
Education
Medical School: Univ Of Mi Med Sch, Ann Arbor Mi 48109
Graduation Year: 1985

Data Provided by:
David Pent, MD FACS
4324 E McDonald Dr
Paradise Valley, AZ
Gender
Male
Education
Medical School: New York Univ
Graduation Year: 1954

Data Provided by:
Sheila Gately Gerbarg, MD
Paradise Valley, AZ
Specialties
Obstetrics & Gynecology
Gender
Female
Education
Medical School: Brown Univ Program In Med, Providence Ri 02912
Graduation Year: 1981

Data Provided by:
Sally T Wareing, MD
11209 N Tatum Blvd
Phoenix, AZ
Specialties
Obstetrics & Gynecology
Gender
Female
Education
Medical School: Vanderbilt Univ Sch Of Med, Nashville Tn 37232
Graduation Year: 1986

Data Provided by:
Victor Kissil, DO
(602) 942-2310
2302 W Greenway Rd
Phoenix, AZ
Business
Deer Valley Ob/Gyn
Specialties
Obstetrics & Gynecology

Data Provided by:
Michael Allen Feinstein, MD
(215) 467-2205
10565 N Tatum Blvd
Paradise Valley, AZ
Specialties
Obstetrics & Gynecology
Gender
Male
Education
Medical School: Jefferson Med Coll-Thos Jefferson Univ, Philadelphia Pa 19107
Graduation Year: 1973

Data Provided by:
J Carlos Diazcadena, MD
Paradise Valley, AZ
Specialties
Obstetrics & Gynecology
Gender
Male
Education
Medical School: Univ Militar Nueva Granada, Bogota, Colombia
Graduation Year: 1986

Data Provided by:
Howard Jay Silverman, MD
5895 E Onyx Ave
Scottsdale, AZ
Specialties
Obstetrics & Gynecology
Gender
Male
Education
Medical School: Washington Univ Sch Of Med, St Louis Mo 63110
Graduation Year: 1979

Data Provided by:
Herbert Saul Rigberg, MD
Phoenix, AZ
Specialties
Obstetrics & Gynecology
Gender
Male
Education
Medical School: Hahnemann Univ Sch Of Med, Philadelphia Pa 19102
Graduation Year: 1964

Data Provided by:
Data Provided by:

Early Warning Signs of Preeclampsia

Provided by: 

By Kathryn Ayers

High blood pressure, fluid retention, and excess protein in the urine all signal the onset of preeclampsia, a condition that affects one in 20 women during the third trimester of pregnancy. While a cause for concern in all cases—about 200,000 women suffer from preeclampsia in the US each year, and it is a leading cause of premature births—only those women who experience large and sudden increases in blood pressure face the threat of severe complications for themselves and their fetuses.

Researchers already knew that changes in the concentrations of two proteins—placental growth factor (PlGF) and soluble fms-like tyrosine kinase 1 (sFit1)—keyed the onset of milder forms of preeclampsia but did not seem to cause the life-threatening levels of high blood pressure seen in severe cases. Their new study in The New England Journal of Medicine names a third factor, soluble endoglin, that seems to kick preeclampsia into overdrive. This protein, which the researchers found in high concentrations in the placentas of women with severe preeclampsia, normally helps maintain the blood vessels, but when it sloughs off the blood vessel walls and combines with sFit1, it actually weakens them.

The study reports that soluble endoglin levels begin to increase markedly two to three months before the onset of preeclampsia and that this increase is usually accompanied by an increased ratio of sFit1 to PlGF. This forewarning should enable researchers to develop a diagnostic test that will allow healthcare providers to counter a major cause of maternal and fetal mortality worldwide.

Author: Kathryn Ayers

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